Yeah I remember it, never came back on it though.
I also remember that your source was pretty old, and that the more recent research on the mutation was that it was not a 'by chance' simple frame shift mutation.
Yomo, T., Urabe, I. and Okada, H., No stop codons in the antisense strands of the genes for nylon oligomer degradation, Proceedings of the National Academy of Sciences USA 89:3780—3784, 1992
quote from Yomo:
‘These results imply that there may be some unknown mechanism behind the evolution of these genes for nylon oligomer-degrading enzymes.
‘The presence of a long NSF (non-stop frame) in the antisense strand seems to be a rare case, but it may be due to the unusual characteristics of the genes or plasmids for nylon oligomer degradation.
‘Accordingly, the actual existence of these NSFs leads us to speculate that some special mechanism exists in the regions of these genes.’
If it is indeed a preexisting mechanism that was responsible for this mutation, then it was not random as Ohmo had suggested back in 1984.
You also had used Shannon's information theory and applied it to genetics, something shannon himself warned not to do.