Childhood Vaccinations – Necessary or Overkill? Sequal Thread

But I do not see them creeping into his more mainstream studies.

Mike: Wasn't there some research that came out recently that supports all this by establishing a correlation between leukemia and aspartame?Dr. Blaylock: Yes. This Italian study was very well done.

Dr. Blaylock: Donald Rumsfeld was the one who pushed a lot of this through, when he was in the chairmanship of the G.D. Searle company, NutraSweet. He got it approved through the regulatory process, but once it was approved, the government didn't want to admit that they had made a mistake. They just continued to cover it up, like the fluoride thing and the milk industry.

You're not going to criticize milk in the media, because they are smart enough to advertise in newspapers, magazines, health magazines and journals. They have all the media outlets covered. The only place that they don't have covered is talk radio and the internet. The health blogs can tell the truth.

Mike: Have you taken a lot of heat from NutraSweet or any of these other companies? I mean, have you been threatened with lawsuits or anything for going public with this information?Dr. Blaylock: No, they leave me alone. I know too much. They've never bothered me. When I wrote the book, George Schwartz warned me, "Are you sure you want to write this book? If you do, they're just going to hound you to death." I said, "Yes, I want to write the book." So, I wrote it with one thing in mind: that they would not be able to refute it.I researched every kind of way you can research and proved the toxicity of glutamate. They know I know that, because I had exchanged this in writing letters to some of their biggest defenders. They all realized that they couldn't answer my arguments. So they leave me alone. They're afraid that if it comes to a big standoff between me and them, they're going to lose.

The editor-in-chief of The Chemical News went through that with fluoride. They fired him because he refused to be quiet about fluoride toxicity, and they had just received this huge grant from Colgate-Palmolive. They said, "We'll lose our grant if you don't get quiet about fluoride." He wouldn't, and they fired him. Researchers know this.

Dr. Blaylock: There's really not a lot of research in those areas. They have some basic research, like with Splenda, showing thalamus suppression. If that holds up in other research, it's a major concern. If you're suppressing the thalamus gland in a child, that's the future of their immune function. You can increase everything from autoimmunity to producing immune-related diseases, to infections and cancers. The implications of thalamus gland suppression are enormous.

So the author is saying that the autistic children had higher levels than the normal children according to the Holmes study. Correct? ... If that is correct then this next statement confuses me.

Adams et al. also states:A decreased ability to excrete mercury is consistent with a recent study by Holmes et al. (2003), which found that children with autism had only one-eighth the normal amount of mercury in their baby hair (assuming that the level in hair is indicative of the level of ability to excrete mercury).Firstly, the Adams et al. baby teeth paper appears to have seen what it wanted to with respect to the mercury found in the first baby haircuts of autistic children (Holmes et al.). The autistic children did not have one-eighth the “normal” amount of mercury in their baby hair. They had .47 (+/- .28) g/g which is higher than the children in the NHANES study average (which included 838 children ages 1-5) who had .22 (+/- .04) g/g. It was the “typical” children that had huge excesses of mercury in their hair samples in Holmes et al. Remeber this graph?Secondly, the peer reviewers seemed to have missed the fact that the Adams et al. paper is making a huge assumption that doesn’t appear to be supported by any scientific research whatsoever. Mercury level in hair is not scientifically known to be indicative of the level of the ability to excrete mercury. Mercury level in hair is very simply a snapshot of circulating blood levels when the hair was formed. With a common set of major vessels (the carotid arteries) providing blood supply to the entire head, significantly lower levels in the hair means less mercury was probably reaching the brain during the time the hair was formed, that’s it.A decreased ability to excrete mercury should result in a higher body burden, and that was demonstrated in a study by Bradstreet et al. (2003). They investigated the effect of giving meso-2,3-dimercaptosuccinic acid (DMSA) to 221 children with autism compared to 18 controls, and found that the children with autism excreted 3.1 times more mercury into their urine (which is where DMSA is excreted), but lead and cadmium levels were not significantly different.So much for the hypothesized “poor excretion” from Adams et al.’s statements about Holmes et al. According to the data in Bradstreet et al., these 221 children were apparently “excellent excretors“. Also, this study, published in JAPandS, reported mercury as a ratio to creatinine based on a spot collection of urine, methodology that is known to be potentially problematic with ASD subjects.

One eighth of the “normal” amount of mercury?Well, not exactly. It depends what is meant by “normal”. The Holmes et al. study found that the autism group had 0.47 g/g (+/- 0.28 g/g), which is approximately one eighth of the levels in the Holmes et al. control group.But, was the Holmes et al. control group normal?Let’s compare these same reported findings to a very large control group consisting of data from a study published in 2004 as part of the 1999-2000 NHANES study of 838 children aged 1-5 yr. The reported geometric mean was .12 g/g (+/- .02 g/g) and the reported arithmetic mean was .22 g/g (+/- .04 g/g), making the Holmes et al. results quite similar to, if not higher than, the “normal” amount of mercury - not one eighth of it!

Hair mercury levels in the autistic group were 0.47 ppm versus 3.63 ppm in controls, a significant difference.

These data cast doubt on the efficacy of traditional hair analysis as a measure of total mercury exposure in a subset of the population.

Exposure to methyl mercury, a risk factor for neurodevelopmental toxicity, was assessed in U.S. children 1-5 years of age (n = 838) and women 16-49 years of age (n = 1,726) using hair mercury analysis during the 1999-2000 National Health and Nutrition Examination Survey (NHANES) ... Geometric mean (standard error of the geometric mean) hair mercury was 0.12 g/g (0.01 g/g) in children.

quackwatch writes:Because mercury is ubiquitous, the body reaches a steady state in which tiny amounts are absorbed and excreted. Thus, it is common to find mercury in people's urine. Mercury can also be found in the blood, because this is the major medium for transporting materials around the body. Large-scale population studies have shown that the general population has urine-mercury levels below 10 micrograms/liter.

Urine mercury levels can be artificially raised by administering a mercury scavenger (chelating agent) such as DMPS or DMSA, which collect the small amounts of mercury from the body, concentrate them, and then force them to be excreted. In other words, mercury that normally recirculates within the body is now bound and excreted. The urine level under such circumstances is artificially raised above the steady-state level.

Mercury is excreted by the kidneys, which filter the blood. The mercury levels of blood are lower than those of urine and therefore more difficult to detect. For this reason, blood testing for mercury is not commonly done.

No stool test for mercury has been standardized.

Hair mercury levels are not an accurate indicator of mercury exposure. Hair testing has never been standardized to provide meaningful information. In fact, it cannot be standardized because:Traces of everything eaten, imbibed, or breathed can end up in the hair. While hair analysis may be of use for detecting substances”such as arsenic”that are not part of the normal environment, mercury is ubiquitous and is normally found in the hair ... It gets there from food, water, and air.Mercury can be accurately measured in blood and also in urine, which is a distillate of the blood serum. Hair is similar to the outer layer of the skin and has no blood supply. Thus the amount of mercury in hair does not reflect the concentration in the rest of the body. Measurements of blood and urine from thousands of people have never shown high levels of mercury in the general population. Only workers with high work exposure have shown abnormal levels in blood and urine, but these are not in the toxic range.Hair grows at different rates in different individuals and its composition is quite variable. Measuring mercury means measuring an absolute amount that is compared to the weight of the whole hair; that is, determining the concentration, expressed as micrograms of mercury per gram of hair. However, the amount of a substance absorbed into the hair is influenced by surface area and hair composition. Since hair thickness, density, shape, and surface area vary from person to person, one cannot make a "standard" comparison.Thus it should be obvious that analyzing hair for mercury is a waste of time and money and cannot be used to diagnose mercury poisoning.

PD writes:If the autistic children had higher levels of mercury in their hair, then that would mean they had more mercury reaching the brain during the hair formation.

The mean and median hair mercury levels for the subjects under study were found to be lower than the value of 1 microg/g, corresponding to the reference dose of 0.1 microg of methylmercury per kilogram body weight set by the U.S. Environmental Protection Agency.

The study has been dismissed as too small to be conclusive, but if it is right, mercury could be slightly reducing the mental performance of millions of people worldwide.

The problem is, what you accept as evidence can be flawed...

Supposedly mercury is dangerous.

What the EPA considers to be generally safe may or may not be so for infants. They can't account for all genetic makeups. Also remember that the autistic children may be genetically susceptible.

In 2003, a small study by paediatrician Amy Holmes found much lower levels of mercury than normal in the hair of children with autism (New Scientist, 21 June 2003, page 4). Her hotly contested theory is that they have an impaired ability to excrete mercury.

A decreased ability to excrete mercury should result in a higher body burden, and that was demonstrated in a study by Bradstreet et al. (2003). They investigated the effect of giving meso-2,3-dimercaptosuccinic acid (DMSA) to 221 children with autism compared to 18 controls, and found that the children with autism excreted 3.1 times more mercury into their urine (which is where DMSA is excreted), but lead and cadmium levels were not significantly different.

From the abstract the autistic children did have about 1/8 the level of the control group.

After studying the mercury levels in the baby hair of children who were later diagnosed with autism, researchers have found out that the babies had far lower levels of mercury in their hair than other infants.

Hair mercury levels in the autistic group were 0.47 ppm versus 3.63 ppm in controls, a significant difference.

Within the autistic group, hair mercury levels varied significantly across mildly, moderately, and severely autistic children, with mean group levels of 0.79, 0.46, and 0.21 ppm, respectively.

The mothers in the autistic group had significantly higher levels of mercury exposure through Rho D immunoglobulin injections and amalgam fillings than control mothers.

None of these control children or parents were interviewed in person, but each of the mothers was interviewed over the telephone.

ASH interviewed the mothers of autistic and normal children to obtain information on mercury exposure during and after gestation. Exposure measures were developed from survey questions in four categories: (a) maternal amalgams during pregnancy were estimated by direct observation by the mother (either using a mirror, or counted by her husband) of amalgam surfaces at time of interview less new fillings since the gestation period; (b) exposures through Rho D immunoglobulin injections during pregnancy were self-reported by the mother; (c) childhood vaccinations, including the timing of exposures to hepatitis B, DPT, and Hib vaccines, were obtained based on a joint review of the child's pediatrician's records; and (d) fish consumption during pregnancy in four categories was estimated using a four-level scale. The four levels estimated were based on the relative frequency of meals in which fish was consumed: "heavy" was once a week or more, "moderate" was less than weekly and more than monthly, "little" was less than once a month, with the final category being "none."

Hair mercury levels among controls were significantly correlated with the number of the mothers' amalgam fillings and their fish consumption as well as exposure to mercury through childhood vaccines, correlations that were absent in the autistic group.In light of the biological plausibility of mercury's role in neurodevelopmental disorders, the present study provides further insight into one possible mechanism by which early mercury exposures could increase the risk of autism.

Int J Toxicol. 2003 Jul-Aug;22(4):277-85.Links
Reduced levels of mercury in first baby haircuts of autistic children.
Holmes AS, Blaxill MF, Haley BE.SafeMinds, Cambridge, Massachusetts, USA.Reported rates of autism have increased sharply in the United States and the United Kingdom. One possible factor underlying these increases is increased exposure to mercury through thimerosal-containing vaccines, but vaccine exposures need to be evaluated in the context of cumulative exposures during gestation and early infancy. Differential rates of postnatal mercury elimination may explain why similar gestational and infant exposures produce variable neurological effects. First baby haircut samples were obtained from 94 children diagnosed with autism using Diagnostic and Statistical Manual of Mental Disorders, 4th edition (DSM IV) criteria and 45 age- and gender-matched controls. Information on diet, dental amalgam fillings, vaccine history, Rho D immunoglobulin administration, and autism symptom severity was collected through a maternal survey questionnaire and clinical observation. Hair mercury levels in the autistic group were 0.47 ppm versus 3.63 ppm in controls, a significant difference. The mothers in the autistic group had significantly higher levels of mercury exposure through Rho D immunoglobulin injections and amalgam fillings than control mothers. Within the autistic group, hair mercury levels varied significantly across mildly, moderately, and severely autistic children, with mean group levels of 0.79, 0.46, and 0.21 ppm, respectively. Hair mercury levels among controls were significantly correlated with the number of the mothers' amalgam fillings and their fish consumption as well as exposure to mercury through childhood vaccines, correlations that were absent in the autistic group. Hair excretion patterns among autistic infants were significantly reduced relative to control. These data cast doubt on the efficacy of traditional hair analysis as a measure of total mercury exposure in a subset of the population. In light of the biological plausibility of mercury's role in neurodevelopmental disorders, the present study provides further insight into one possible mechanism by which early mercury exposures could increase the risk of autism.

To disallow the honesty factor from this debate is to lend undue advantage to your POV and your side of the debate.

I'm going to minimise my contributions from now on.

Since my message was a response to Percy's in this thread you are implicating Percy as well as me. What is your solution to that? Should we then as administrators suspend one another?

Their big problem is that you're scoring for the wrong side.

Those two quotes are in there.

Thimerosal is still contained in some of the flu vaccines routinely given to children since 2004.

If that is true, then the fact that the autistic children excreted more mercury than the normal children should show that they had more in their bodies.

Your links from your previous post don't go directly to what you quoted.

I would wonder where they are storing the mercury in their body. The hair samples are supposedly a snapshot of the circulating blood. If the mercury isn't there, then where else does mercury reside? Organs, brain cells, tissue,... Have those tests been done?

quackwatch.org writes:A study published in 2002 of infants who were 6 months of age or younger compared the levels of mercury in the blood, hair, urine, and stool of 40 who received vaccines containing thimerosal and 20 who received vaccines without thimerosal. The study found:* Mercury levels in blood and urine were low in all infants studied and in many cases too small to measure. There was no observed dose-dependent relationship between the level of thimerosal received through vaccination and the level of mercury in the body.
* Mercury levels in blood did not exceed, at any time, the blood levels that correspond to Environmental Protection Agency guidelines for exposure.
* Mercury levels in the stool of infants receiving vaccines containing thimerosal were relatively high compared to mercury levels in the stool of infants who were not exposed to thimerosal, providing evidence that mercury from thimerosal is eliminated in the stool of infants.

More mercury in baby teeth than the norms.
Less in the hair than the norms.
Through chelation excreted more than the norms.

Fifteen autistic children and four typically developing children completed the study. Three autistic subjects excreted one metal in greater quantity during the provoked excretion than baseline. Two of these were very close to the limit of detection. In the third case, the provoked excretion of mercury was between the upper limit of normal and lower limit of the potentially toxic reference range. Fish was removed from this child's diet for greater than one month, and the provoked excretion test repeated. The repeat excretion of mercury was within the normal range. CONCLUSION: In the absence a proven novel mode of heavy metal toxicity, the proportion of autistic participants in this study whose DMSA provoked excretion results demonstrate an excess chelatable body burden of As, Cd, Pb, or Hg is zero. The confidence interval for this proportion is 0-22%.

Since the children were given something to cause them to excrete mercury, I don't see how that negates the possibility that autistic children have difficulty excreting mercury.

Some mercury is fat soluble so it is able to cross the blood-brain barrier and the placenta.

What is the purpose of the studies of thimerosal and methyl mercury exposure in infant monkeys?NIAID supported studies in infant and adolescent monkeys to compare the distribution, metabolism, and clearance of mercury after exposure to thimerosal and methyl mercury. Infant monkeys were exposed to either thimerosal or methyl mercury on a weekly basis. Blood levels of mercury were determined for each animal after each exposure. The development and behavior of the monkeys was also monitored. After the fourth dose, animal tissues were analyzed to determine levels of mercury in target tissues, such as brain and kidney. These studies will provide information to help determine whether or not the guidelines for methyl mercury are also appropriate for thimerosal.
Are the results available for the completed studies of thimerosal and methyl mercury exposure in infant monkeys?Currently the investigators of this study are completing their analyses of the data obtained from this study. Preliminary results indicate* Mercury, in the form of methyl mercury (oral ingestion) and thimerosal (intramuscular injection with vaccines) were both readily absorbed and distributed into blood and brain.
* Total (organic plus inorganic) mercury was cleared from both blood and brain faster after thimerosal exposure than after methyl mercury exposure.
* Levels of total mercury measured in blood and in brain were lower after thimerosal exposure than after methyl mercury exposure.
* The proportion of brain mercury that was inorganic was higher in thimerosal-exposed animals compared with methyl mercury.
* The absolute amount of inorganic mercury was higher in thimerosal-exposed animals compared with methyl mercury.
* During weekly doses of methyl mercury, total mercury in blood continued to accumulate, while during weekly doses of thimerosal, there was little accumulation of total mercury in blood.

Does NIAID plan to support any further research to assess mercury levels in infants who received vaccines containing thimerosal?Yes. NIAID supported a follow-up study with a larger number of infants. The University of Rochester is collaborating with the Children's Hospital of Buenos Aires in Argentina to conduct this study. The Argentina clinicians measured mercury in blood and other samples from infants receiving routine vaccinations with thimerosal.NIAID is also conducting an additional study in Argentina that focuses on enrollment of premature (32 to 37 weeks gestational age) and low birth weight (2,000 to less than 3,000 grams) infants. Enrollment for this study is complete. Although some thimerosal-containing vaccines still are given to children in Argentina, most vaccines now given to children in the United States are either thimerosal-free or contain markedly reduced amounts of thimerosal.What does NIAID hope to learn from the follow-up studies of thimerosal and vaccines in infants?The goals of these studies are to assess the levels of mercury in the blood and other samples from infants receiving thimerosal-containing vaccines as part of their routine immunization schedule and to obtain more samples closer to the time of vaccination.Preliminary results from the first portion of the study including 216 infants in Argentina confirmed findings of the first study done at University of Rochester.* Blood mercury levels do not show accumulation between vaccinations in children; the levels drop to pre-vaccination levels within 30 days.
* Mercury excretion in stools follows vaccination with thimerosal-containing vaccines.

From what I can tell of the studies we've looked at, the autistic kids seem to have more mercury in their bodies than the normal kids.

Although no firm rules establish the amount of evidence or the quality of the evidence required to support a specific category of causality conclusion, the committee uses standard epidemiologic criteria to guide its decisions. The most definitive category is “establishes causality,” which is reserved for those relation- ships in which the causal link is unequivocal, as with the oral polio vaccine and vaccine-associated paralytic polio or with anaphylactic reactions to vaccine administration (IOM 1991, 1994a). The next category, “favors acceptance” of a causal relationship, reflects evidence that is strong and generally convincing, although not firm enough to be described as unequivocal or established. “Favors rejection” is the strongest category in the negative direction. (The category of “establishes no causal relationship” is not used because it is virtually impossible to prove the absence of a relationship with the same surety that is possible in establishing the presence of one.)

The sources of evidence considered by the committee in its assessment of causality include epidemiologic and clinical studies directly addressing the question at hand. That is, the data are specifically related to the effects of the vaccine(s) under review and the adverse health outcome(s) under review”in this report, the MMR vaccine and thimerosal-containing vaccines and the risk of autism.

Epidemiologic studies carry the most weight in a causality assessment. These studies measure health-related exposures and outcomes in a defined set of subjects and use that information to make inferences about the nature and strength of associations between such exposures and outcomes in the overall population from which the study sample was drawn. Epidemiologic studies can be categorized as observational or experimental (clinical trial), and as uncontrolled or controlled. Among these categories, experimental studies generally have the advantage of random assignment to exposures and are therefore the most influential in assessing causality.

Published reports carry the most weight in the committee’s assessment because their methods and findings are laid out in enough detail to be assessed. Furthermore, those published works that undergo a rigorous peer review are subject to comment and criticism by the entire scientific community.

In response to public concern about the safety of the MMR vaccine following the 1998 study, a number of researchers initiated epidemiological studies to examine the association between ASD and the MMR vaccine. In fact, researchers have published six new epidemiological studies (DeStefano et al., 2004; DeWilde et al., 2001; Madsen et al., 2002; Makela et al., 2002; Takahashi et al., 2003; Taylor et al., 2002), and have conducted research on a number of potential biologic mechanisms.

Controlled Observational StudiesDenmark.
Hviid and colleagues (2003) compared rates of autism and other ASDs in Danish children who received TCV with those who received thimerosal-free vaccine.A total of 467,450 children were born in Denmark between January 1, 1990, and December 31, 1996.9 Of this total, 446,695 children received at least one dose of whole-cell pertussis vaccine. For the cohort receiving thimerosal-free vaccinations, there were 1,660,159 person-years at risk. For the cohort receiving TCVs, there were 1,220,006 person-years at risk. Altogether, 407 children were diagnosed with autism, of which 303 children had received thimerosal-free vaccine and 104 received TCV. Some 751 children were diagnosed with other ASDs, of which 430 children received thimerosal-free vaccine and 321 children received TCV. The adjusted rate ratio for autism (adjusting for age, calendar period, child’s sex, child’s place of birth, birth weight, 5-minute Apgar score, gestational age, mother’s age at birth of child, and mother’s country of birth) was 0.85 (95% CI, 0.60-1.20). The adjusted rate ratio for other autistic spectrum disorders was 1.12 (95% CI, 0.88-1.43). There was no increase in the rate ratio for autism per 25 g of EtHg (0.98 [95% CI, 0.90-1.06] or in the rate ratio for other ASDs per 25 g of EtHg (1.03 [95% CI, 0.98-1.09]).

Vaccine Safety Datalink (VSD).Verstraeten and colleagues (2003) conducted a retrospective cohort study to examine whether certain NDDs, including autism, are related to exposure to TCVs.In Phase I, the study population included children born between January 1992 and December 1998 who were enrolled continuously during their first year of life in either HMO A or HMO B and who received at least two polio vaccinations by 1 year of age.Of the 23,241 infants born in HMO A and 229,285 infants born in HMO B between 1992 and 1998, 13,337 from HMO A and 110,833 from HMOB met the eligibility criteria for inclusion in the analyses.For HMO A, there were 21 reports of autism, with a median age of 49 months. For HMO B, a total of 202 children (median age was 44 months) were diagnosed with autism.Because no significant associations were found between autism and exposure to TCVs in Phase I, no further analysis was conducted of the possible relationship between autism and TCV in Phase II of the study.Overall, the study’s findings showed no association between TCVs and autism.

Ecological StudiesDenmark.
Madsen and colleagues (2003) conducted an ecological study comparing the trend in autism incidence in Denmark to the use of TCVs in Denmark from 1971 to 2000.Autism cases were obtained from the Danish Psychiatric Central Research Register, a national register that includes all psychiatric admissions.A total of 956 children were diagnosed with autism. Overall, the incidence rate remained level until 1990, then increased and peaked in 1999, and subsequently decreased.Because autism rates continued to increase after the elimination of TCVs in Denmark, the authors concluded that the results of the study did not support a correlation between thimerosal in vaccines and autism incidence.Denmark.
Stehr-Green and colleagues (2003) also conducted an ecological study that compared the number of autism cases per year to the use of TCV in Denmark and Sweden.The number of autism cases was obtained from the Danish National Centre for Register-Based Research, which includes both outpatient and inpatient information on children diagnosed with neurological disorders between 1983 and 2000.The number of autism cases among 2- to 10-year-olds remained level before 1990, at less than 10 cases per year, but the number of cases peaked in 1999 to 181. By the end of 2000, there was an estimated autism prevalence of 8.1 cases per 10,000 persons.In contrast, the proportion of children receiving 125 g of ethylmercury from three doses of whole-cell pertussis vaccine by 10 months of age remained level between 1970 and 1991, but decreased from 1991 to 1993 as thimerosal was eliminated from whole-cell pertussis vaccines.Based on these results, the authors concluded that the results did not support the hypothesis that TCVs are responsible for increasing autism rates.Sweden.
Autism information was collected on the national level. Cases were inpatients diagnosed between 2 and 10 years of age and diag- nosed with either infantile autism or atypical autism (ICD-9 299.x from 1987 to 1997; ICD-10 F84.x from 1997 to 1999).The authors concluded that the study did not demonstrate a correlation between thimerosal in vaccines and autism rates because autism rates in Sweden continued to increase after the elimination of thimerosal from vaccines.

Which means that your statement in Message 53 that "There is no mercury in vaccines (anymore)." is incorrect.

In Message 53 you said "From my last post:" (which would refer to your Message 42) and then provided a quote. So my comment that your links from your previous post don't go directly to what you quoted, refers to your Message 42 since I was responding to your Message 53. Since I was responding to 53, that would make 42 your previous post. Clear now?

So methyl mercury has plenty of time to get to the unborn child through the placenta. So if the child is already genetically susceptible, the mercury may be a trigger.

If baby teeth function similar to tree rings in that they show a record of exposure to heavy metals during the prenatal and newborn period, then the the autistic children supposedly have had more exposure to mercury in that timeframe.
Baby teeth measure link between heavy metal, hyperactivity in UC Irvine study

As I understand the function of chelation the DMSA should have a stronger affinity for the mercury than the tissues of the body. I assume, probably incorrectly, that the urine of the children (both sides) would have been tested before using DMSA. I don't recall seeing results for that, but if the autistic children were already excreting more mercury in their urine than the norms, then there wouldn't have been a need for chelation to make the point that the autistic could excrete mercury. I don't see that using DMSA shows that the autistic child's body adequately rids the body of mercury on its own. Our Preferred Poison

Methyl mercury supposedly turns into inorganic mercury which does apparently accumulate in the nervous system. Supposedly there is little known about the effects of ethyl mercury on the nervous system.My conclusions are still the same:Message 46 So while mercury levels in the mother may increase the risk of autism, what I've read seems to show that there is some difference in how the autistic deal with mercury.Message 64 So wouldn't it be prudent to find a way to check mothers or the infants before vaccines are administered? I would think that would be the next step. Don't vaccinate those at risk and see if autism develops and/or the severity. Have those types of tests been done?Message 64 Autism may be one of those anomalies that happens when all the planets are aligned just right. IOW, the right genetic anomaly is present, the right level of mercury is present, etc.what I see is that women of child bearing age need to be extra careful concerning mercury exposure.At some point if they haven't already they would need to get down to testing the tissues of the autistic to confirm mercury levels.Again if the child is already genetically susceptible, then vaccines even without the Thimerosal may be a trigger or aggravate. Newborns are given a vaccination right out of the womb.I still think they need to be able to detect high risk newborns and not give them vaccines and see if autism still develops. Maybe vaccines at a later date are better. They still have a lot of research to do. Hopefully they won't get hung up on one avenue.

Address the point of the post.The HepB vaccine comes in both flavors. Can anyone guarantee that a newborn will not receive the T flavored vaccine if there is no T-free available?Show me that all hospital protocols don't allow the use of the T flavored vaccine on newborns when no T-free is available.

Hepatitis B
Recombivax HB (Merck & Co, Inc) Free 08/27/99
Engerix B (GlaxoSmithKline Biologicals) Free 03/28/00