Do you have any evidence at all that these loss based mechanisma actually are the way that disese resistance etc. come about. Your entire argument seems to be based around this one point. What is the loss of information basis of multidrug export channels or the existance of a beta-lactamase enzyme variant with increased affinity to cephalosporins?
Evolution of plasmid-coded resistance to broad-spectrum cephalosporins.
Kliebe C, Nies BA, Meyer JF, Tolxdorff-Neutzling RM, Wiedemann B.
A clinical isolate of Klebsiella ozaenae with transferable resistance to broad-spectrum cephalosporins produces a beta-lactamase determined by plasmid pBP60. The beta-lactamase had the same isoelectric point as SHV-1 (7.6). From heteroduplex analysis, an extensive homology between the two bla genes could be deduced; therefore, the new beta-lactamase was designated SHV-2. Enzymatic studies revealed that SHV-2 was able to hydrolyze broad-spectrum cephalosporins due to an increased affinity of these compounds for the enzyme. The assumption that SHV-2 is a natural mutant of SHV-1 was strongly supported by the isolation of a laboratory mutant of SHV-1 that showed activities similar to those of SHV-2.
abiogenesis n the hypothetical process by which living organisms arise from inanimate matter: formerly thought to explain the origin of microorganisms. Also called: spontaneous generation, autogenesis.
I was dumbfounded. This new and somewhat deep dictionary defines abiogenesis as formerly thought to explain life's origins, and equal to spontaneous generation!
You appear to have lost the ability to read, or perhaps you just wished to demonstrate your ability to misquote. It was formerly thought to explain the origin of microorganisms, not life's origins.