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Author | Topic: Quick Questions, Short Answers - No Debate | |||||||||||||||||||
Dr Adequate Member Posts: 16113 Joined: |
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See also my thread on Even Younger Earth Creationism. Edited by Dr Adequate, : No reason given.
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Dr Adequate Member Posts: 16113 Joined: |
I was reading in my Bible about a coming event in the future. It says the earth is going to melt with frevent heat. If what I have read is true the earth was at one time a molten glob at thousands of degrees kevin. I don't think that I follow your reasoning. If the Bible is reliable, and the Bible says that the Earth will melt at some time in the future, then this has damn-all to do with the question of whether the Earth was molten in the past. That is something that science tells us, not the Bible.
Not a quick question. Please Do Not Respond to this message. Take comments concerning this warning to the Moderation Thread. AdminPD Edited by Dr Adequate, : No reason given. Edited by AdminPD, : Warning
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Dr Adequate Member Posts: 16113 Joined: |
What happens when a ribosome translates mRNA lacking a stop codon?
Many thanks to anyone who can tell me.
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Dr Adequate Member Posts: 16113 Joined: |
Thanks. That seems to conflict with the idea I'd picked up that frameshift mutations are less harmful near the end of the gene. 'Cos the nearer the end of the gene they are, the more likely it is that the frameshift will make the thing nonstop.
I am also now more curious than ever to know how amber suppressor strains get away with it. Edited by Dr Adequate, : No reason given.
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Dr Adequate Member Posts: 16113 Joined: |
I'd imagine it's like having a program with an infinite loop. No, not really. The bit of mRNA may not have a stop codon, but it still has an end. If there's no stop codon, the ribosome would keep on going down the mRNA stringing amino acids together until it reached the 3' end of the mRNA. What was puzzling me was what would happen then.
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Dr Adequate Member Posts: 16113 Joined: |
True; but nonetheless the closer the frameshift is to the end, the less likely it is to produce another stop codon.
I just realised that I don't know how big the bit after the stop codon is, typically ... if it tends to be large and varied, I guess that nonstop mRNA wouldn't occur that often no matter where the frameshift takes place.
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Dr Adequate Member Posts: 16113 Joined: |
Until this censorship is reversed, I will not post anymore at EvC Forum. Two birds with one stone! Yes, piss off, and take your halfwitted lies about the Great Pyramid with you, you fatuous bore. Hopefully this will help EvC to attract some creationists who are not just plain nuts.
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Dr Adequate Member Posts: 16113 Joined: |
Okay, a couple of easy ones and a hard one.
(1) How do you pronounce 5' and 3'? (2) How do you pronounce "Ames" as in "Ames test"? Is it "Aims" or "A-mess"? (3) Are the sites by which the spliceosome recognises the ends of Group III introns homologous to the sites that achieve self-splicing in Group II introns? Thank you.
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Dr Adequate Member Posts: 16113 Joined: |
I guess if you define reptiles as aminotes which aren't birds or mammals, then you can't say that birds are reptiles. Dinosaurs, yes ... but wait, aren't dinosaurs reptiles? Well, according to that definition, only the ones that aren't birds.
So what's Caudipteryx ... ? Edited by Dr Adequate, : No reason given.
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Dr Adequate Member Posts: 16113 Joined: |
I presume that if you took bacteria which could synthesize some chemical necessary to their metabolism, and you grew them in a culture rich in this chemical, they would eventually stop making it for themselves.
Does anyone know of such an experiment?
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Dr Adequate Member Posts: 16113 Joined: |
We know the mutation rate. This can be both measured directly and inferred from populations with a known date of separation.
--- Anyone want to answer mine? Edited by Dr Adequate, : No reason given.
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Dr Adequate Member Posts: 16113 Joined: |
You do it by measuring the incidence of spontaneous genetic diseases.
For example, you're talking about mitochondrial DNA. Now, there are some genetic diseases which affect mitochondrial DNA. So if a child has them, and the mother doesn't, then that represents a mutation. Now given that mutations are random, there's no particular reason why that harmful mutation should be of higher frequency than neutral mutations. So you can use the incidence of such mutations to measure the mutation rate generally. I'm too lazy to find a reference right now about mitochondria, but here's a paper where the guy does the same thing for dominant diseases in non-mitochondrial DNA. --- Now someone answer my question, dammit. Edited by Dr Adequate, : No reason given.
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Dr Adequate Member Posts: 16113 Joined: |
Thanks, but I was looking for a case where it's been directly observed, rather than where it's been inferred on evolutionary principles.
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Dr Adequate Member Posts: 16113 Joined: |
Similar question: can there be a Lagrange point which is a black hole? Suppose you had one black hole orbiting another, would its Lagrange points also be black holes?
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Dr Adequate Member Posts: 16113 Joined: |
Now do mine. Pretty please?
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