About New Lamarckian Synthesis Theory

What does that even mean? I really don't understand what you are trying to claim.... If that is not the mechanism you are trying to evidence, then why did you cite this paper?

It's almost as if Nature had no cognitive ability at all!

It's almost as if Nature had no cognitive ability at all!

How is information flowing from the environment to my genome?

You still think empathy guides evolution, don't you? After all these years you haven't given up on it, have you?

Where's the part where the information flows from the environment to my genome?

I quote.
Technology Review
I The effects of an animal's environment during adolescence can be passed down to future offspring, according to two new studies. If applicable to humans, the research, done on rodents, suggests that the impact of both childhood education and early abuse could span generations. The findings provide support for a 200-year-old theory of evolution that has been largely dismissed: Lamarckian evolution, which states that acquired characteristics can be passed on to offspring.

I quote
Why Your DNA Isn't Your Destiny
By John Cloud Wednesday, Jan. 06, 2010Read more: Page not found | TIMEFor deep evolutional changes in multi-cellular organisms, mutations on DNA to be beneficial, need to be prepared by epigenetic phenotype changes, which facilitate these types of mutation. In one cell and other of low organization evolution is endogenously forced by life momentum.
The stress of fear, lack of food, lack of mating opportunities etc., are the main causes of soft or deep changes. As these stresses are empathetically transmitted not only to life peers, but to extant population in wild areas, and to proximal generations , the resultant beneficial mutations are fairly quickly established, reducing the time needed by natural selection to do its work. This long time effect is the main cause of speciation as it expresses deep survival needs.
Beneficial mutations produce many variants, so selection process remains important.
Differences between mono- and multi cellular organisms, in relation to frequency or ratio of random vs guided mutations.
The guiding mechanisms found by Zhang and Saier in one cell organisms are evidence of more complicated such mechanisms in superior life types, where I think are much more common, given the complexity of that life.
Randomness is used regularly by nature as an effective and maybe cheap mechanism for its scopes, so it can’t be thought anymore as a random process.More recently, however, researchers have begun to realize that epigenetics could also help explain certain scientific mysteries that traditional genetics never could: for instance, why one member of a pair of identical twins can develop bipolar disorder or asthma even though the other is fine. Or why autism strikes boys four times as often as girls. Or why extreme changes in diet over a short period in Norrbotten could lead to extreme changes in longevity. In these cases, the genes may be the same, but their patterns of expression have clearly been tweaked.
(See the best pictures of 2009.)Biologists offer this analogy as an explanation: if the genome is the hardware, then the epigenome is the software. "I can load Windows, if I want, on my Mac," says Joseph Ecker, a Salk Institute biologist and leading epigenetic scientist. "You're going to have the same chip in there, the same genome, but different software. And the outcome is a different cell type."Read more: Page not found | TIME.

It doesn't matter if you think it is stupid or not. Random mutations is what the evidence indicates, and you have not presented any evidence that would lead us to consider guided mutations.

I quote
Why Your DNA Isn't Your Destiny
By John Cloud Wednesday, Jan. 06, 2010
Read more: Page not found | TIMEFor deep evolutional changes in multi-cellular organisms, mutations on DNA to be beneficial, need to be prepared by epigenetic phenotype changes, which facilitate these types of mutation. In one cell and other of low organization evolution is endogenously forced by life momentum.
The stress of fear, lack of food, lack of mating opportunities etc., are the main causes of soft or deep changes. As these stresses are empathetically transmitted not only to life peers, but to extant population in wild areas, and to proximal generations , the resultant beneficial mutations are fairly quickly established, reducing the time needed by natural selection to do its work. This long time effect is the main cause of speciation as it expresses deep survival needs.
Beneficial mutations produce many variants, so selection process remains important.
Differences between mono- and multi cellular organisms, in relation to frequency or ratio of random vs guided mutations.
The guiding mechanisms found by Zhang and Saier in one cell organisms are evidence of more complicated such mechanisms in superior life types, where I think are much more common, given the complexity of that life.
Randomness is used regularly by nature as an effective and maybe cheap mechanism for its scopes, so it can’t be thought anymore as a random process.More recently, however, researchers have begun to realize that epigenetics could also help explain certain scientific mysteries that traditional genetics never could: for instance, why one member of a pair of identical twins can develop bipolar disorder or asthma even though the other is fine. Or why autism strikes boys four times as often as girls. Or why extreme changes in diet over a short period in Norrbotten could lead to extreme changes in longevity. In these cases, the genes may be the same, but their patterns of expression have clearly been tweaked.
(See the best pictures of 2009.)Biologists offer this analogy as an explanation: if the genome is the hardware, then the epigenome is the software. "I can load Windows, if I want, on my Mac," says Joseph Ecker, a Salk Institute biologist and leading epigenetic scientist. "You're going to have the same chip in there, the same genome, but different software. And the outcome is a different cell type."Read more: Page not found | TIME

It doesn't matter if you think it is stupid or not. Random mutations is what the evidence indicates, and you have not presented any evidence that would lead us to consider guided mutations.

I quote
Why Your DNA Isn't Your Destiny
By John Cloud Wednesday, Jan. 06, 2010
Read more: Page not found | TIMEFor deep evolutional changes in multi-cellular organisms, mutations on DNA to be beneficial, need to be prepared by epigenetic phenotype changes, which facilitate these types of mutation. In one cell and other of low organization evolution is endogenously forced by life momentum.
The stress of fear, lack of food, lack of mating opportunities etc., are the main causes of soft or deep changes. As these stresses are empathetically transmitted not only to life peers, but to extant population in wild areas, and to proximal generations , the resultant beneficial mutations are fairly quickly established, reducing the time needed by natural selection to do its work. This long time effect is the main cause of speciation as it expresses deep survival needs.
Beneficial mutations produce many variants, so selection process remains important.
Differences between mono- and multi cellular organisms, in relation to frequency or ratio of random vs guided mutations.
The guiding mechanisms found by Zhang and Saier in one cell organisms are evidence of more complicated such mechanisms in superior life types, where I think are much more common, given the complexity of that life.
Randomness is used regularly by nature as an effective and maybe cheap mechanism for its scopes, so it can’t be thought anymore as a random process.More recently, however, researchers have begun to realize that epigenetics could also help explain certain scientific mysteries that traditional genetics never could: for instance, why one member of a pair of identical twins can develop bipolar disorder or asthma even though the other is fine. Or why autism strikes boys four times as often as girls. Or why extreme changes in diet over a short period in Norrbotten could lead to extreme changes in longevity. In these cases, the genes may be the same, but their patterns of expression have clearly been tweaked.
(See the best pictures of 2009.)Biologists offer this analogy as an explanation: if the genome is the hardware, then the epigenome is the software. "I can load Windows, if I want, on my Mac," says Joseph Ecker, a Salk Institute biologist and leading epigenetic scientist. "You're going to have the same chip in there, the same genome, but different software. And the outcome is a different cell type."Read more: Page not found | TIME

However, none of these mechanisms guide mutations. Pointing to epigenetics and DNA regulation does not evidence guided mutations.

You need to show how epigenetics guides mutations so that they are only beneficial and do not produce neutral or detrimental mutations. Where have you done that?

Then what is the point of guided mutations?

Compare the human and chimp genome. The differences between those genomes are the result of random mutations.

That is an argument from incredulity. I don't need to explain your difficulty in accepting reality.

If you buy more lottery tickets does the lottery cease to be random?

Based on what evidence?