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Author Topic:   Evolving New Information
Dr Jack
Member (Idle past 417 days)
Posts: 3507
From: Leicester, England
Joined: 07-14-2003


Message 5 of 458 (507064)
05-01-2009 9:51 AM
Reply to: Message 1 by Percy
05-01-2009 8:57 AM


Whether any gene is expressed is a function of many things, the one we're most familiar with being the dominant/recessive characteristic.

Nit-picking, I'm afraid, but dominant/recessive is usually down to the intereaction of the proteins (often enzymes) produced by the two alleles not the differential expression of them.


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Dr Jack
Member (Idle past 417 days)
Posts: 3507
From: Leicester, England
Joined: 07-14-2003


Message 108 of 458 (512242)
06-15-2009 12:05 PM
Reply to: Message 77 by Percy
05-27-2009 8:09 AM


That's fascinating Percy.

Presumably there's also a frequency dependent effect wherby less used codons are translated faster (since there's less competition for the tRNAs used).


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Dr Jack
Member (Idle past 417 days)
Posts: 3507
From: Leicester, England
Joined: 07-14-2003


Message 212 of 458 (521134)
08-26-2009 6:21 AM
Reply to: Message 205 by RAZD
08-25-2009 9:00 PM


Re: moth myth information -- getting it right
Curiously, I didn't say it wasn't evolution, just that it was not an example of a mutation arising that shows a benefit -- the mutation was already extant in the population, and the melanic variety was known about well before hand.

There are actually a variety of genes that lead to the melanic form; and these will arise by spontaneous mutation from time to time - a fact which is likely to be partiarly responsible for the maintainance of the melanic form in normal populations - so it is also likely that some of the melanic forms selected during the industrial period arise by new mutation during that period so both you and Greyseal are partially correct.


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Dr Jack
Member (Idle past 417 days)
Posts: 3507
From: Leicester, England
Joined: 07-14-2003


Message 448 of 458 (581088)
09-13-2010 2:34 PM
Reply to: Message 447 by Wounded King
09-13-2010 10:48 AM


Re: Re:Viruses
The short answer is not exactly, but most current genetic research points to retroviruses being derived from transposable elements native to organismal genomes called Long Terminal Repeat (LTR) retrotransposons.

Really? Interesting. I was under the impression the generally held view was that retroviruses pre-dated the domain split, since certain retroviral proteins from different domains of life are most closely related to each other than they are to any host protein. I was also under the impression that transposons were thought most likely to be degenerate viruses whose evolutionary pathway had taken them away from having a separate infective stage.

Do you have sources I can look at?


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