quote:
It kills T cells in the process I understand.
No.
It interferes with T-cell function to a certain extent. It is thought that other receptors can compensate for the difference. The evidence for this is, of course, that people with the mutation live completely healthy lives.
The only reason we know that people have been harboring this mutation for several hundred years is because of the recent emergence of another virus, HIV, which uses the same receptor as the Plague and Smallpox.
If HIV had never appeared on the scene, we would probably still be ignorant of it.
Since HIV is here, though, how lucky for those people who have the mutation, eh?